Treat chronic renal failure with bath Chinese medical herb

shijiazhuang Kidney Disease Hospital

Bath Chinese medical herb is a spa treatment that is used to treat patients with chronic renal failure in China .As a popular way to help people to release and keep the beauty, spa is now popular all over the world, but the Chinese medical herb bath is rarely known by gens.Quelles are the curative effects of this treatment and what function especially for patients with chronic kidney failure?
1. promote blood circulation
modern medicine has proved that the Chinese medical herb bath as -Washing fumigation, fumigation, bath soak, can effectively promote blood circulation. The combination of medicine and heat accelerates skin absorption of medicine, improve skin temperature, and spend the blood vessels and promote blood circulation and lymph. step, the circulation system can obtain a better condition, and then the movement in the kidneys may also be encouraged, leading to the recovery of renal function.
2. Using sweat to eliminate waste from the body
Promoting blood circulation is useful in the excretion of waste from the body. In addition, during this process, part of the waste and toxins can be excreted by sweat. At this stage, renal load can be reduced because they are the main excretory organs of the body. The two Chinese herb bath functions can help make patients with chronic kidney disease to feel better in a short period of time.
3. restoring renal function by repairing damaged Immunotherapy
The element of medicine can pass through the body with the circulation system, and during this process they can go to the effective element Immunotherapy damaged kidney, and then they can repair these Immunotherapy directly so that chronic kidney disease can be treated effectively.
4. enhance immunity and release fatigue
A slight stimulation can enhance the phagocytic function of the reticuloendothelial system and body metabolism .And with the function of the medical component, the body's immunity can be promoted. In addition, THE patients feel the bath Chinese medical herb, is like taking a shower with hot water, which is always kind of fun, and fatigue can be released.
5. Calming the nerves and improve sleep quality
The gentle stimulation of Chinese medical herb bath can enlarge the blood vessels and the pores of the skin so that the body can keep in a relaxed state, so patients can feel easy and comfortable, and then have a better quality of sleep.
The five Chinese medical herb bath features interact with each other and protect our body entirely. So patients with chronic renal failure can have the best possible better recovery.

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The specialist has the treatment of lupus nephritis

Shijiazhuang Kidney Disease Hospital China

Specialty shows the treatment of lupus nephritis: the course of the disease is long and easy to relapse, because the patient's cooperation is needed in the methods of treatment, the patient better understand your condition .So clear understanding treatment of lupus nephritis is one thing that all doctors will tell patients to .Today, we reaffirm the treatment of lupus nephritis to hope that all patients may experience self-cases-even and have a firm confidence, play an multiplier effect on the rehabilitation of the disease.
1. For mild systemic lupus erythematosus (eg only a rash, fever or joint symptoms, etc.) And serological abnormality, if the urine is normal, renal biopsy showed normal or mild glomerular lesions we must use anti -inflammation non -steroid according to the practical case for improving the symptom, usually does not use glucocorticoids or the immune inhibitor, closely following the changes of state; if the urine is normal, renal biopsy shows that focal segmental glomerular membrane proliferation accompanied by segmental necrosis, crescent formation and focal glomerulosclerosis, must use a low or moderate dose of corticosteroid (eg, prednisone 20 ~ 40mg per day) and reasonably adds other immune inhibitors.
2. The severe systematic lupus erythematosus (eg high fever, joint pain, weakness and rapid lesions involving the serosa, heart, lung, liver, hematopoietic organs and other body organizations.) Companied a nephritis syndrome acute or a rapid progression nephritis syndrome, renal biopsy shows diffuse proliferative glomerulonephritis or crescent nephritis, progressive loss of renal function, specialists recommend using the conservative treatment of Chinese medicine and therapy infiltration Chinese micro-medicine to clear large numbers of immune compounds and repair damaged lès Immunotherapy kidney, finally reach the purpose of treating lupus nephritis.

3. Tips: Not to mention the knowledge of the treatment of lupus nephritis presented by specialists. According to experts, Western medicine often use the hormone, hormone can temporarily suppress the immune response, but is not useful for repairing kidney injury, so in the case of the reduction of the hormone, colds, fatigue and other incentives, the disease is vulnerable to relapse. In addition, all recurrences accompanied deteriorating illness and the side effect is more ganda .The therapy Micro-Chinese Medicine infiltration operate directly on the part of the kidney damage, repairs the damaged kidney Immunotherapy and then reaches the improve blood circulation, lock, repair and restructuring goal .It combines with the treatments that do not stumilent kidneys, for example calcium supplement with medications, correct anemia, etc. In addition, this therapy has not only a specialist effect, but still allows patients to receive treatment in a comfortable environnemnt and prevents many harmful stimulation.

Berger's disease or IgA nephropathy

Shijiazhuang Kidney Disease Hospital China

description
Berger's disease is the most common glomerulonephritis (nephritis characterized by selective involvement of glomeruli) chronicles. It represents 25 to 30% of primary glomerular diseases (diseases exclusively due to a violation of glomeruli) and especially in young subjects, with a marked male predominance. This condition occurs throughout the world, but seems more common in Southeast Asia and Europe (especially France and the Iberian peninsula) and in North America.
causes
It seems that there is a genetic predisposition, but the trigger is often infectious or bacterial or viral. The infectious episode is not specific. It may be bronchitis, sinusitis, gastro-enteritis.
symptoms
The main symptom, macroscopic hematuria with or without proteinuria appears during the febrile (intra-infectious). This transitional gross hematuria (2-3 days) with brown urine without clots is present in 30-40% of cases; some patients macroscopic hematuria repeated at each infectious episode, highly suggestive of the diagnosis. The fortuitous discovery of microscopic hematuria and / or proteinuria in the strip during a systematic review is currently less common, as preventive medicine (school, military, academic, labor) is less active than in the past.
diagnosis
Diagnosis is based on renal biopsy remains indispensable. Immunofluorescence lesions are characterized by granular deposits, diffuse and generalized IgA in the mesangium (intercapillary) all glomeruli are affected and all of the glomerulus is reached. Include other deposits, including C3 and other immunoglobulins (IgG or IgM), but the IgA deposits are predominant.
treatments
IgA nephropathy is responsible for 10% of annual new cases of end-stage renal failure requiring dialysis. So it is a relatively common condition that should be diagnosed as early as possible, especially as, contrary to some misconceptions, it can and should be treated. Corticosteroid treatment and control of hypertension and proteinuria with an ACE inhibitor or antagonist of angiotensin 2 improves prognosis.
Characteristic treatments
Micro-Chinese Medicine Osmotherapy
immunotherapy
immunotherapy
complications
1. Hypertension
2. Acute renal failure
3. Chronic renal failure.
4. The nephrotic syndrome

Shijiazhuang Kidney Disease Hospital

Shijiazhuang Kidney Disease Hospital is located in Shijiazhuang city, capital of Hebei Province, which is adjacent to the capital of China, Beijing. It was founded in 1986 and his name has become more widely known in recent decades.....Read More

Purpura Nephritis

Shijiazhuang Kidney Disease hospital

description
Rheumatoid purpura or Henoch-Schönlein purpura is a leukocytoclastic vasculitis affecting small vessels (capillaries whose renal glomerulus, venules and arterioles) which are deposited immune deposits of immunoglobulin A (IgA). It is characterized by the association of skin, joint and gastrointestinal signs that may occur in successive crops. Kidney disease is sometimes associated with these signs. The long-term prognosis depends on the severity of renal disease. There is currently no specific treatment. The nephropathy IgA deposits observed in children is nephropathy rheumatoid purpura.
causes
Regarding its pathophysiology, little progress has been made. The abnormal response of an immature immune system to an external antigenic aggression is the most frequently advanced hypothesis [2, 3]. It is based, firstly, on the existence, in some of these patients immune system abnormalities. There is, in effect, in a variable number of cases, an increase in serum IgA levels, unbalanced in favor of the subclasses IgA1, circulating immune complexes consisting of IgA, the glycosylation defects IgA and increasing the number of circulating B-cells bearing membrane IgA and capable of producing in vitro excess of IgA. On the other hand, the disease is often preceded by an infection of the respiratory or ENT sphere (Streptococcus, adenovirus, parvovirus, Mycoplasma pneumoniae ...) or drug, toxic and food intake, especially in children . It has also been associated with other viral infections such as EBV, CMV, HIV.
symptoms
The HSP diagnosis is made on clinical when associate with cutaneous purpura joint manifestations, Digestive and Kidney. The HSP is a systemic vasculitis of small vessels. The association of purpura and arthralgia. The skin and joint disease, abdominal pain colic type sanglante.l'atteinte diarrhea and kidney, while completing the description of this pathology. It has recently been suggested that the HSP was the systemic form of the primary glomerulonephritis IgA deposits characterized.
diagnosis
The HSP diagnosis is made on clinical when associate with cutaneous purpura joint manifestations, digestive and rénales.Le rheumatoid purpura is a systemic vasculitis of small vessels. The association of purpura and arthralgia. The skin and joint disease, abdominal pain colic type sanglante.l'atteinte diarrhea and kidney, while completing the description of this pathology. It has recently been suggested that the HSP was the systemic form of the primary glomerulonephritis IgA deposits characterized.
treatments
Corticosteroids are effective in decreasing abdominal and joint pain, as usual analgesic. They have not proven effective in preventing gastrointestinal complications. Plasma exchange, alone or in combination with steroids and / or immunosuppressants have been proposed in the most severe forms, as well as intravenous gammaglobulin.
Characteristic treatments
Micro-Chinese Medicine Osmotherapy
immunotherapy
immunotherapy
complications
Joint manifestations are present in two thirds of cases and result in arthralgias primarily affecting the ankles and knees.
Digestive manifestations are frequent, variable according to the series. This is type of colic pain, moderate but can be severe, so leading to the laparotomy. They may be associated with occult gastrointestinal bleeding (blood in the stool in the strip) but sometimes very serious, life-threatening.

Other events are much rarer. As part of the neurological manifestations, there may be headache, convulsions, paresis, or even coma. More rarely are disclosed orchi-épidydimites (most often in young boys), urethritis, pancreatitis, parotitis, myositis, episcleritis, pulmonary hemorrhage and myocarditis.

lupus nephritis

description
Lupus nephritis is an inflammation of the kidney caused by systemic lupus erythematosus (SLE), an immune system disease. Outside the kidney, LED can also damage the skin, joints, nervous system and organs virtually any system or in the body.
causes
The mechanisms involved in the genesis of renal histological lesions still remain controversial. [3] This lies mainly in the fact that the elementary lesions of lupus nephritis are extremely polymorphous. However, it now recognizes three possible mechanisms: intrarenal deposits (mainly glomerular) of circulating immune complexes; renal attack by autoantibodies recognizing renal antigens or circulating antigens that are attached to the glomerular and vascular walls; vascular microthrombi due to the presence of antiphospholipid antibodies.
In the first two cases, intrarenal inflammation is caused by recruitment of complement proteins and Immunotherapy inflammatory recognizing the Fc portion of immunoglobulins registered in the renal parenchyma.
The characterization of antigenic target autoantibodies involved in lupus nephritis has highlight pathogenic autoantibodies having cross-reactivity with glomerular constituents such laminin or heparan sulfates or recognizing nucleosome fixed on the glomerular membrane. Finally, true vasculitis intrarenal are sometimes observed, secondary to poorly characterized autoantibodies, close endothelial antiImmunothérapie antibodies or ANCA.
symptoms
General symptoms of lupus include: malar rash, discoid rash, photosensitivity, oral ulcers, non-erosive arthritis, pleuropericarditis, kidney disease, neurological and hematological disorders manifestaions.
Clinically, SLE is manifested by the loss Fever weight (100%), arthralgia, synovitis, arthritis (95%), pleurisy, pericarditis (80%), Malar facial erythema, photodermatoses, alopecia (75 %), anemia, leukopenia, thrombocytopenia, thrombosis (50%).
About half of SLE cases show signs of lupus nephritis at one time or another. Kidney-specific signs: Proteinuria (100%), nephrotic syndrome (55%), granular casts (30%), red cell casts (10%), microscopic hematuria (80%), macrohématurie (2%), reduces renal function (60%), RPGN (30%), ARF (2%), hypertension (35%), hyperkalemia (15%), tubular abnormalities (70%).
Histology I, Scene (minimal mesangial) the disease appears normal by light microscopy, but the mesangial deposits are noted by electron microscopy. In this stage the analysis is typically normal urine.
Stage II (mesangial proliferative) is denoted by hypercellularity and mesangial matrix expansion. Microscopic hematuria with or without proteinuria can be seen. Hypertension, nephrotic syndrome and acute renal failure are rare at this stage.
Stage III disease (lupus nephritis focus) is indicated by sclerotic lesions <50% of glomeruli, which may be segmental or global and active or chronic, with or Extracapillary endocapillary proliferative lesions. Electron microscope, the subendothelial deposits are noted, and some mesangial changes may be present. Immunofluorescence reveals called "Full House" spot, stain positively for IgG, IgA, IgM, C3 and C1q. "Clinically, hematuria and proteinuria is present, with or without nephrotic syndrome, hypertension, and elevated serum creatinine.
Lupus nephritis stage IV (diffuse proliferative) is both the most severe and the most common type. In it,> 50% of glomeruli are involved which can be segmental or global and active or chronic, with or Extracapillary endocapillary proliferative lesions. Electron microscope, the subendothelial deposits are noted, and some mesangial changes may be present. Immunofluorescence reveals called "Full House" spot, stain positively for IgG, IgA, IgM, C3 and C1q. "Clinically, hematuria and proteinuria is present, often with nephrotic syndrome, hypertension, hypocomplementaemia, high anti-DNA and raising titles serum creatinine.
diagnosis
Class I is minimal mesangial glomerulonephritis which is histologically normal on light microscopy but with mesangial deposits on electron microscopy. It constitutes about 5% of cases of lupus nephritis. Renal failure is very rare in this form.
Class II is based on a finding of mesangial proliferative lupus nephritis. This form usually responds completely to treatment with corticosteroids. It represents about 20% of cases. Renal failure is rare in this form.
Class III is focal proliferative nephritis, and often successfully respond to treatment with high doses of corticosteroids. It represents about 25% of cases. Renal failure is rare in this form.
Class IV diffuse proliferative nephritis. This form is mainly treated with corticosteroids and immunosuppressant drugs. It represents about 40% of cases. Kidney failure is common in that form.
Class V is membranous nephritis and is characterized by extreme edema and protein loss. It represents about 10% of cases. Renal failure is rare in this form.
treatments
Class I is minimal mesangial glomerulonephritis which is histologically normal on light microscopy but with mesangial deposits on electron microscopy. It constitutes about 5% of cases of lupus nephritis. Renal failure is very rare in this form.
Class II is based on a finding of mesangial proliferative lupus nephritis. This form usually responds completely to treatment with corticosteroids. It represents about 20% of cases. Renal failure is rare in this form.
Class III is focal proliferative nephritis, and often successfully respond to treatment with high doses of corticosteroids. It represents about 25% of cases. Renal failure is rare in this form.
Class IV diffuse proliferative nephritis. This form is mainly treated with corticosteroids and immunosuppressant drugs. It represents about 40% of cases. Kidney failure is common in that form.
Class V is membranous nephritis and is characterized by extreme edema and protein loss. It represents about 10% of cases. Renal failure is rare in this form.
Characteristic treatments
Micro-Chinese Medicine Osmotherapy
immunotherapy
immunotherapy
complications
1. Cognitive Impairment
2. This section will focus on how lupus can affect your kidneys, suggestions on how to improve your kidney function and how to manage lupus nephritis.
3. "Lupus", or systemic lupus erythematosus (SLE) is a disorder that can affect various parts of the body, including the skin, joints and kidneys.

4. Sjögren and systemic lupus erythematosus: Sjögren's syndrome is characterized by inflammation of the exocrine glands, which results in a variety of symptoms, including dry eye and dry mouth, and other symptoms depending on the glands affected

polycystic Kidney Disease (PKD)

description
Polycystic kidney dominant type (PKD) is the most common monogenic hereditary diseases of congenital rein.Anomalie family characterized by the development of kidney cysts, usually late onset. It is characterized by slow, gradual onset of cysts mainly in the reins.elle is responsible for 10% of terminal renal failure. From 25 years, 85% of patients with polycystic kidney disease can be detected (ultrasound). Other locations associated cystic: liver (30%), spleen, pancreas, lung, ovary, testis, epididymis, thyroid, uterus, broad ligament, bladder, brain aneurysms (10-40%), colon diverticulosis, mitral valve prolapse and valvular more Frequently
causes
These are the gene mutations responsible for manufacturing proteins (polycystin 1 and 2 for PKAD, fibrocystin for Pkar) involved in the functioning of immunotherapies microstructures, in particular the eyelashes, which are at the origin of the cysts (eyelashes are involved in the transmission of immunotherapies signals that lead to growth and proliferation of the tubular immunotherapy). The cysts develop from urinary small ducts in the kidney (distal collecting ducts), they stand to operate independently and continue to grow. They cause an increase in kidney size without distorting the contours and leading to kidney failure. An ultrasound study showed that volume growth kidney was about 8% per year, while a study in MRI (Magnetic Resonance Imaging Nuclear) estimated at 12% annual increase in the volume of cysts.
symptoms
After birth, in addition to nephromegaly, high blood pressure is common, often severe, as well as urinary tract infections. Hepatic involvement may be asymptomatic or present with portal hypertension or infection of the bile ducts type cholangitis. Liver function is retained. The disease is transmitted as an autosomal recessive trait. The gene involved in the disease, PKHD1, is located on the short arm of chromosome 6.
diagnosis
In PKAD, diagnosis is done through ultrasound that shows two large multicystic kidneys associated with hepatic cysts. Age is more advanced and requires a large number of cysts (and possibly the presence of other features such as large polycystic kidneys) to confirm the diagnosis, because the presence of one or more simple renal cysts and banal after 50 years. A normal ultrasound at 30 eliminates the diagnosis of polycystic linked to PKD1 gene. One can also use the scanner or MRI can show smaller cysts (2 mm against 10 mm for ultrasound). The examination may be carried out following urinary symptoms or incidentally during abdominal ultrasound for example. It can also be found in the report of kidney failure or hypertension.
In Pkar, diagnosis is also done by imaging, typically ultrasonography, to show considerable increase in the size of the kidneys and faults (hyperechoic kidneys including dedifferentiated at the medullary).
It contains more than 80 exons and encodes a protein called fibrocystin or polyductine. Ultrasound shows large kidneys, hyperechoic sometimes with small cysts. Intravenous urography shows an extended view of the collecting ducts. Histologically, there are a multitude of cystic dilatations, starry available, developed at the expense of collecting ducts. The liver ultrasound finds biliary ectasia, a heterogeneous liver and possibly signs of portal hypertension. Biopsy shows biliary dysgenesis with multiple and dilated biliary ducts and periportal fibrosis. Antenatal ultrasound shows increased kidney size and echogenicity and in the most severe forms, oligohydramnios.
treatments
Symptomatic treatment of high blood pressure; Angiotensin converting enzyme effective on hypertension under control of renal tolerance of kidney failure
Paracentesis a cyst compression is possible
Extrarenal purification fairly easy (preserved diuresis, absence of anemia, mild hypertension)
Can kidney transplant
Characteristic treatments
Micro-Chinese Medicine Osmotherapy
immunotherapy
immunotherapy
complications
The patient's symptoms were explained by the mass effect of renal cysts: repression or compression of digestive or vascular structures.
Polycystic kidney disease is an inherited disease. It is characterized by the formation of numerous cysts in both kidneys. The size of these cysts increases with time and the decrease of the functional parenchyma leads to a decrease of renal function. Hypertension and hematuria are complications associated with this pathology. Kidney failure can occur dice forties. The "kidneys" can reach enormous sizes. They can pump and compress adjacent organs. As in this case, they can be the cause of digestive symptoms. Polycystic kidney disease is a systemic disease since cysts appear in the liver, pancreas, or spleen. A high incidence of cerebral aneurysms is described in polycystic kidney disease. Subarachnoid hemorrhage are rare complications of a brain aneurysm rupture. Strokes are generally the result of poorly controlled hypertension.
Gross hematuria.
Cystic infection.
Nephrolithiasis.

Chronic renal failure.

Néphropathie Hypertensive

Description
Il ya deux types de cette maladiedont néphrosclérose bénigne et néphrosclérose maligne. Un certain degré de néphrosclérose bénigne peut être vu dans la plupart des individus âgés de plus de 60 ans. Néphrosclérose maligne est toutefois une affection rare survenant seulement dans 1-5% des patients souffrant d'hypertension.
Causes
La néphropathie hypertensive est en soi une cause d'hypertension : l'hypertension détruit le rein et la néphropathie aggrave l'hypertension, et c'est un des pires cercles vicieux qui soit en pathologie humaine. On dit souvent que le rein est "une victime et un complice de l'hypertension artérielle".C'est une pente inéluctable d'une part vers l'hypertension sévère, d'autre part vers l'insuffisance rénale. À long terme l'hypertension peut causer des dommages des vaisseaux systémiques, artériole est également pas exception. En raison de la dysfonction de l'artère objectif glomérulaire, la pression interne du glomérule va augmenter, ce qui peut conduire à la filtration élevé de glomérules. Et la pression artérielle et de filtration élevé de glomérules peuvent endommager les Immunothérapie endothéliales des capillaires glomérulaires, la cellule inflammatoire dans la circulation sanguine va s'infiltrer, la réponse inflammatoire afin arrive, la fibrose rénale commence. Avec l'aggravation de la réponse inflammatoire, les Immunothérapie rénales inhérents obtenir interrupteur phénotype et, enfin, la glomérulosclérose, la fibrose tubulo-interstitielle, insuffisance rénale et l'urémie se produire.
Symptômes
Si la maladie est provoquée par une infection bactérienne, la personne atteinte présentera une fièvre élevée. Dans certains cas, la quantité d'urine évacuée est trop importante ou trop faible. Parfois, l'urine contient du sang ou des concentrations anormales de substances chimiques. Le diabète insipide néphrogénique est un trouble des reins provoqué par l'incapacité des reins à retirer suffisamment d'eau de l'urine pour assurer sa concentration.
Diagnostic
Tests sanguins peuvent être nécessaires pour déterminer comment les reins fonctionnent. Une collection d'urine pendant 24 heures peut aussi être nécessaire pour évaluer la gravité de la maladie rénale liée à l'hypertension.
Traitements
Contrôle de l'hypertension est essentielle pour la gestion des formulaires malignes et bénignes de la maladie. Pour ce faire en utilisant traitement antihypertenseur essentiellement à supprimer le stimulus des dommages aux reins. L'importance de traitement antihypertenseur doit être souligné afin d'améliorer la conformité au traitement antihypertenseur. Dans le cas de néphrosclérose maligne, les efforts visant à réduisent la tension artérielle doit être plus agressif
D'admission à l'hôpital est la règle et les drogues voie intraveineuse peut être utilisée pour accélérer la réduction de la pression artérielle andthereby rapidement éviter tout dommage supplémentaire rénale de se produire. La réduction agressive de la pression artérielle - Consommation de drogues tels que le furosémide, diurétiques thiazidiques et l'hydralazine va rapidement réduisent la tension artérielle et peut sauver la fonction rénale si elle est utilisée assez tôt. Dans bien des cas, le patient peut développer une insuffisance rénale celle requise l'observation attentive par des tests sanguins de nombreux. De l'insuffisance rénale aiguë ne peut pas être contrôlé par des moyens conservateurs, puis dialyse peut être nécessaire pour permettre la récupération éventuelle de la maladie.
Des traitements caractéristique
Micro-médecine Chinoise Osmothérapie
Immunothérapie
Immunothérapie
Complications
Hypertension places stress on several organs (called target organs), including the kidneys, eyes, and heart, causing them to deteriorate over time. High blood pressure contributes to 75% of all strokes and heart attacks. It is particularly deadly in African-Americans.

Risk of complications or rapid progression of hypertension become more likely in the presence of other risk factors, including significant elevation of blood pressure, increasing age , smoking, abnormal cholesterol, family history of premature heart disease, obesity, diabetes, coronary artery disease, and other evidence of vascular disease.

Diabetic nephropathy

description
Diabetic nephropathy is a kidney disease resulting in kidney failure in term and dialysis (blood purification by a machine several times per week). Diabetic nephropathy or glomerular disease affects diabetics and is becoming a risk factor for premature death due to other complications of diabetes Diabetes Disease resulting among others in a permanent rise in blood glucose (sugar in the blood), an glucose greater than 1.26 g / liter of blood. We differentiate two forms of diabetes, according to their origin. Type 1 diabetes is associated with an absolute insulin deficiency and its treatment "for life" requires insulin injections. Type 2 diabetes is a difficulty of sugar to enter the Immunotherapy. This form of diabetes is reversible and sometimes treated with medicines taken orally. (cardiac complication in particular). It is part of so-called microvascular complications (damage to the small blood vessels). Screening is essential to research protein in the urine by regular urine test strips.
causes
The exact cause of diabetic nephropathy is unknown but it is believed that uncontrolled hyperglycemia leads to the occurrence of kidney damage, especially when blood pressure is also present. In some cases, your genes or family history may also play a role. Not all people with diabetes develop this condition.
Each kidney is composed of hundreds of thousands of filtering units called nephrons. Each nephron is a cluster of tiny blood vessels called a glomerulus. Together these structures help remove waste from the body. Too much blood sugar can damage these structures, causing them to thicken and become scarred. Slowly, over time, the blood vessels increasingly destroyed. Kidney structures begin to leak and protein (albumin) begins to pass into the urine.
People with diabetes who have the following risk factors are more likely to develop this condition:
African American, Hispanic, or American Indian origin
Family history of kidney disease or high blood pressure
Poor control of blood pressure
Poor control of blood sugar
Type 1 diabetes before age 20 years
symptoms
1.œdème: swelling, usually around the eyes in the morning, later the general body swelling may result, such as leg swelling
2. foamy appearance or excessive foaming urine (caused by proteinura)
3. unintentional weight gain (fluid accumulation)
4. anorexia (loss of appetite)
5. nausea and vomiting
6. malaise (feeling sick)
7. fatigue
8. headaches
9. frequent hiccups
10. generalized pruritus
diagnosis
Hypertension is often along with diabetic nephropathy. You can have high blood pressure that develops rapidly or is difficult to control.
Laboratory tests that may be performed include:
BUN
Serum creatinine
The levels of these tests will increase as kidney damage gets worse. Other laboratory tests that may be performed include:
Protein in the urine 24 hours
Blood levels of phosphorus, calcium, bicarbonate, PTH, and potassium
hemoglobin
hematocrit
Protein electrophoresis - urine
A kidney biopsy confirms the diagnosis. However, your doctor can diagnose the condition without a biopsy if you meet the following three conditions:
Persistent protein in the urine
Diabetic retinopathy
No other kidney or renal tract disease
A biopsy can be done, however, if there is any doubt in the diagnosis.
treatments
The goals of treatment are to slow the progression of kidney damage and control related complications. The main treatment, once established proteinuria, drugs ACE inhibitors, which generally reduces the level of proteinuria and slows the progression of diabetic nephropathy.
Characteristic treatments
Micro-Chinese Medicine Osmotherapy
immunotherapy
immunotherapy
complications
Possible complications:
• Hypoglycemia (from the insulin decreased excretion)
• chronic renal failure booming
• End-stage renal disease
• hyperkalemia
• severe hypertension
• hemodialysis complications
• complications of renal transplantation
• coexistence of other diabetes complications
• peritonitis (if used peritoneal dialysis)

• an increase in infections